Today's HEADLINE! S'pore rewrite table tennis history! Feng TianWei and Wang YueGu, the stunners of China world leading players!!! Amazing Feng TianWei as the comeback queen. Final result: S'pore-China 3-1! Congratulation to S'pore team to end the 18-year domination of China team in women's table tennis!!!
In Men's event, Germany Timo Boll showed his great comeback by winning the first match against Ma Long and fought well in the 4th game against Ma Lin although he lost at last. Germany lost 1-3 to China, China defend the title for the 5th time in row.
Sunday, May 30, 2010
Tuesday, May 25, 2010
Adams-Stokes syndrome
It is sudden, transient episode of syncope, occasionally featuring seizures. It is named after two Irish physicians, Robert Adams (1791–1875) and William Stokes (1804–1877).
Before an attack, patient may become pale, heart rhythm pause temporally, and collapse may follow. Normal period of unconsciousness is 30 seconds. If seizures are present, there will be twitching after 15–20 seconds. Breathing is normal throughout the attack. On recovery, patient becomes flushed as the blood pumped from pulmonary into systemic circulation which has become dilated because of hypoxia.
NB: As with any syncopal episode that results from a cardiac dysrhythmia, the faints do not depend on the patient's position. If they occur during sleep, the presenting symptom may simply be feeling hot and flushed on waking.
Diagnosis: History taking, observed paleness prior to attack and flushing, ECG - assystolia and ventricular fibrillation during the attacks.
Cause is loss of cardiac output due to cardiac asystole, heart block, Lev's disease or ventricular fibrillation. The resulting lack of blood flow to the brain is responsible for the faint.
Treatment: isoproterenol (Isuprel) and epinephrine (adrenaline). Surgical - insertion of pacemaker.
Prognosis: untreated - 50% mortality within 1 year of 1st episode. Good prognosis after treatment.
http://en.wikipedia.org/wiki/Adams-Stokes_syndrome
Before an attack, patient may become pale, heart rhythm pause temporally, and collapse may follow. Normal period of unconsciousness is 30 seconds. If seizures are present, there will be twitching after 15–20 seconds. Breathing is normal throughout the attack. On recovery, patient becomes flushed as the blood pumped from pulmonary into systemic circulation which has become dilated because of hypoxia.
NB: As with any syncopal episode that results from a cardiac dysrhythmia, the faints do not depend on the patient's position. If they occur during sleep, the presenting symptom may simply be feeling hot and flushed on waking.
Diagnosis: History taking, observed paleness prior to attack and flushing, ECG - assystolia and ventricular fibrillation during the attacks.
Cause is loss of cardiac output due to cardiac asystole, heart block, Lev's disease or ventricular fibrillation. The resulting lack of blood flow to the brain is responsible for the faint.
Treatment: isoproterenol (Isuprel) and epinephrine (adrenaline). Surgical - insertion of pacemaker.
Prognosis: untreated - 50% mortality within 1 year of 1st episode. Good prognosis after treatment.
http://en.wikipedia.org/wiki/Adams-Stokes_syndrome
Wednesday, May 19, 2010
Differentiating angina pectoris (AP) and myocardial infarction (MI)
This is my first medical post!
Angina pectoris and myocardial infarction are two serious diseases that are most commonly interchanged. Early recognition of angina pectoris may prevent the condition from developing into myocardial infarction.
In AP, there is pain due to decreased blood and oxygen supply to the heart. Heart tissue goes back to normal when there is enough oxygen supply.
There are stable and unstable angina. Unstable angina can lead to myocardial infarction (these two are known as acute coronary syndrome).
In MI, the tissue actually dies. Heart tissue never return to normal even oxygen is restored. However, there are ischemic part of tissue which there is low oxygen supply, not died tissue. So we can save those "sick" tissue by reperfusion (open those arteries) as soon as possible.
In few words, AP, reversible. MI, irreversible, died tissue.
References:
http://www.sh.lsuhsc.edu/fammed/OutpatientManual/Angina.htm
http://www.earlysymptoms.net/blog/myocardial-infarction-vs-angina-pectoris/
http://www.123angina.com/angina-pectoris/what-is-the-difference-between-angina-pectoris-and-myocardial-infarction-2
Angina pectoris and myocardial infarction are two serious diseases that are most commonly interchanged. Early recognition of angina pectoris may prevent the condition from developing into myocardial infarction.
In AP, there is pain due to decreased blood and oxygen supply to the heart. Heart tissue goes back to normal when there is enough oxygen supply.
There are stable and unstable angina. Unstable angina can lead to myocardial infarction (these two are known as acute coronary syndrome).
In MI, the tissue actually dies. Heart tissue never return to normal even oxygen is restored. However, there are ischemic part of tissue which there is low oxygen supply, not died tissue. So we can save those "sick" tissue by reperfusion (open those arteries) as soon as possible.
In few words, AP, reversible. MI, irreversible, died tissue.
References:
http://www.sh.lsuhsc.edu/fammed/OutpatientManual/Angina.htm
http://www.earlysymptoms.net/blog/myocardial-infarction-vs-angina-pectoris/
http://www.123angina.com/angina-pectoris/what-is-the-difference-between-angina-pectoris-and-myocardial-infarction-2
Sunday, May 16, 2010
Tutorial : Hide/Encrypt posts in Blogger
Just visited my friend's blog, noticed that there are encrypted texts. So I search for the info, this is what I got.
Try click the following link,
Show encrypted text
Try click the following link,
Show encrypted text
(password: blogger)
Encryption/Decryption site: http://www.vincentcheung.ca/jsencryption/
More info: http://www.vincentcheung.ca/jsencryption/instructions.html
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